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Shae Of Wicked Antler

Shae

Shae is a very gentle loving Akita. Shae has made a fine addition to our line up. 


We are super pleased with her and all the love she gives to everyone that is around her 

Embark DNA Test Results

MATERNAL HAPLOTYPE

 

A2

SHAE OF WICKED ANTLER’S HAPLOGROUP

A2 is a very ancient maternal line. Most likely it was one of the major female lines that contributed to the very first domesticated dogs in Central Asia about 15,000 years ago. Some of the line stayed in Central Asia to the present day, and frequently appear as Tibetan Mastiffs and Akitas. Those that escaped the mountains of Central Asia sought out other cold spots, and are now found among Alaskan Malamutes and Siberian Huskies. This lineage is also occasionally found in several common Western breeds, such as German Shepherds and Labrador Retrievers. Curiously, all New Guinea Singing Dogs descend from this line. These are an ancient and very interesting breed found in the mountains of Papua New Guinea. Unfortunately, they are now endangered. They are closely related to the Australian dingo, so you could say its cousins are dingos! This line is also common in village dogs in Southeast and East Asia. Unlike many other lineages, A2 did not spread across the whole world, probably because it did not have the opportunity to hitch its wagon to European colonialism - or because these dogs just prefer hanging out in mountains, tundras, islands, and other hard-to-reach places!

A39

SHAE OF WICKED ANTLER’S HAPLOTYPE

Part of the A2 haplogroup, this haplotype occurs most commonly in Akitas and Maltese.

DEGENERATIVE MYELOPATHY

 ClearTEST RESULT

Shae Of Wicked Antler has two healthy alleles at SOD1 and is unlikely to develop DM due to mutations in this gene.

Degenerative Myelopathy
(SOD1A)CONDITIONSOD1GENE NAMEGGCLEARGACARRIERAAAT RISKRecessiveINHERITANCE TYPE

DESCRIPTION

A disease of mature dogs, this is a progressive degenerative disorder of the spinal cord that can cause muscle wasting and gait abnormalities. Affected dogs do not usually show signs until they are at least 8 years old, where the first signs of neural degeneration appear in the nerves that innervate the hind limbs. You may notice your dog scuffing the tops of his or her hind paws, or walking with a hesitant, exaggerated gait. In advanced cases, lower motor neurons are also affected leading to weakness or near-paralysis of all four legs and widespread muscle wasting. Given the advanced age at the time of onset, the treatment for DM is aimed towards making your dog comfortable in his or her old age and includes lifestyle changes and physical therapy. SOD1 codes superoxide dismutase, an enzyme important in neutralizing free radicals and reactive oxygen species, both of which are produced as a byproduct of cell metabolism. If not neutralized, these are injurious to the cell and will cause premature cell death. The first system to show effects of this is the nervous system given the highly specialized and delicate nature of these cells. Please note that these mutations are reported to have incomplete penetrance: that is, while a dog with two copies of this mutation has a much greater chance of developing DM than a dog with one copy of the mutation, or none at all, other genetic and environmental factors will also contribute to whether your dog develops DM.

Genetic Health Conditions

 

Genetic Health Conditions

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A genetic health condition indicates a genetic mutation that increases the risk that an animal develops a specific disease.

Not At Risk

Good news! Shae Of Wicked Antler did not test positive for any of the genetic conditions that Embark screens for.

Not A Carrier

Good news! Shae Of Wicked Antler is not a carrier for any of the genetic conditions that Embark tests for.

Good news! Shae Of Wicked Antler tested clear for 7 other common genetic conditions that Embark tests for. 

Other Conditions

 

>Shae Of Wicked Antler is clear of 165 other genetic conditions that Embark tests for. 

Coat Color

 

Coat Color

SEE DETAILSE Locus (MC1R)No dark hairs anywhere (ee)K Locus (CBD103)Not expressed (kyky)A Locus (ASIP)Not expressed (ayay)D Locus (MLPH)Not expressed (DD)B Locus (TYRP1)Likely black colored nose/feet (BB)Saddle Tan (RALY)Not expressed (NN)M Locus (PMEL)No merle alleles (mm)Other Coat Traits

Other Coat Traits

SEE DETAILSFurnishings (RSPO2) LINKAGELikely unfurnished (no mustache, beard, and/or eyebrows) (II)Coat Length (FGF5)Likely short or mid-length coat (GG)Shedding (MC5R)Likely heavy/seasonal shedding (CC)Coat Texture (KRT71)Likely straight coat (CC)Hairlessness (SGK3)Very unlikely to be hairless (NN)Hairlessness (FOXI3) LINKAGEVery unlikely to be hairless (NN)Oculocutaneous Albinism Type 2 (SLC45A2) LINKAGELikely not albino (NN) 

Other Body Features

 

Other Body Features

SEE DETAILSMuzzle Length (BMP3)Likely medium or long muzzle (CC)Tail Length (T)Likely normal-length tail (CC)Hind Dewclaws (LMBR1)Unlikely to have hind dew claws (CC)Blue Eye Color (ALX4) LINKAGELess likely to have blue eyes (NN)Back Muscling & Bulk, Large Breed (ACSL4)Likely normal muscling (CC)Body Size

Body Size

SEE DETAILSBody Size (IGF1)Larger (NN)Body Size (IGFR1)Larger (GG)Body Size (STC2)Larger (TT)Body Size (GHR - E195K)Larger (GG)Body Size (GHR - P177L)Larger (CC)Performance

Performance

SEE DETAILSAltitude Adaptation (EPAS1)Normal altitude tolerance (GG)Genetic Diversity

Genetic Diversity

SEE DETAILSCoefficient Of Inbreeding24%MHC Class II - DLA DRB1High DiversityMHC Class II - DLA DQA1 and DQB1High Diversity