Wicked Antler Kennels ( wickedantlerkennels.com ) is super excited to introduce our newest member to our clan. Quinn is a monster of a Akita with a heart made of gold and as big as the moon. He has taken to Akira & her sister Shika and is there protector. Katashi, Takara & Xena are not total sure of him yet but are getting use to him.
Quinn's color is superb, the eyes are just beautiful, he loves to be hugged. He always has a very happy attitude and just loves the attention that he gets. Although he is still not sure of what to do with hard boiled eggs yet, to play with them or eat them.
Quinn's build is very massive and is big boned. His paws are almost the size of my palm. When he stands up on his hind legs the top of his head is at my nose and paws on my shoulders.
Hes going to make one hell of Sire and can not wait to see the puppies he throws.
Any questions just call us at 573-397-1150 or visit our web site wickedantlerkennels.com, Don't forget to like & share our face book page Wicked Antler Kennels.
We have put this picture in here so you might get a better idea of what some of the puppies with this coat coloring might look like.
Through Quinn’s mitochondrial DNA we can trace his mother’s ancestry back to where dogs and people first became friends. This map helps you visualize the routes that his ancestors took to your home. Their story is described below the map.
A1a is the most common maternal lineage among Western dogs. This lineage traveled from the site of dog domestication in Central Asia to Europe along with an early dog expansion perhaps 10,000 years ago. It hung around in European village dogs for many millennia. Then, about 300 years ago, some of the prized females in the line were chosen as the founding dogs for several dog breeds. That set in motion a huge expansion of this lineage. It's now the maternal lineage of the overwhelming majority of Mastiffs, Labrador Retrievers and Gordon Setters. About half of Boxers and less than half of Shar-Pei dogs descend from the A1a line. It is also common across the world among village dogs, a legacy of European colonialism.
Part of the large A1a haplogroup, this rare haplotype occurs in dogs with Asian ancestry.
C is a relatively rare paternal lineage. The dog populations which bear C are a disparate bunch. The Akita and Shiba Inu are Japanese breeds, the former of which seems to have roots in the Jomon population of hunter-gatherers which were present in the islands of Japan before the ancestors of the modern Japanese arrived. The New Guinea Singing Dog, Samoyed, and Alaska Malamute are all disparate breeds that also represent the C lineage. One village dog from Peru also bore this lineage. This wide distribution and diversity suggest C is not a recently expanded lineage. It likely represents a canid lineage which diversified sometime around the Last Glacial Maximum, when the dogs of Siberia and Oceania split off and went their separate ways.
Part of the C haplogroup, this haplotype occurs most commonly in Akitas and Alaskan Malamutes.
Quinn has two healthy alleles at SOD1 and is unlikely to develop DM due to mutations in this gene.
Good news! Quinn tested clear for 1 genetic conditions that are common in his breed.
(SOD1A)CONDITIONSOD1GENE NAMEGGCLEARGACARRIERAAAT RISKRecessiveINHERITANCE TYPE
A disease of mature dogs, this is a progressive degenerative disorder of the spinal cord that can cause muscle wasting and gait abnormalities. Affected dogs do not usually show signs until they are at least 8 years old, where the first signs of neural degeneration appear in the nerves that innervate the hind limbs. You may notice your dog scuffing the tops of his or her hind paws, or walking with a hesitant, exaggerated gait. In advanced cases, lower motor neurons are also affected leading to weakness or near-paralysis of all four legs and widespread muscle wasting. Given the advanced age at the time of onset, the treatment for DM is aimed towards making your dog comfortable in his or her old age and includes lifestyle changes and physical therapy. SOD1 codes superoxide dismutase, an enzyme important in neutralizing free radicals and reactive oxygen species, both of which are produced as a byproduct of cell metabolism. If not neutralized, these are injurious to the cell and will cause premature cell death. The first system to show effects of this is the nervous system given the highly specialized and delicate nature of these cells. Please note that these mutations are reported to have incomplete penetrance: that is, while a dog with two copies of this mutation has a much greater chance of developing DM than a dog with one copy of the mutation, or none at all, other genetic and environmental factors will also contribute to whether your dog develops DM.
These clinical traits are valuable to your veterinarian and can inform the clinical decisions and diagnoses they make.
A genetic health condition indicates a genetic mutation that increases the risk that an animal develops a specific disease.
Good news! Quinn did not test positive for any of the genetic diseases that Embark screens for.
Good news! Quinn is not a carrier for any of the genetic diseases that Embark tests for.
>Quinn is clear of 167 other genetic diseases that Embark tests for. Click here to see them all!
E Locus (MC1R)No dark mask or grizzle (Ee)K Locus (CBD103)More likely to have a patterned haircoat (kyky)A Locus (ASIP)Fawn Sable coat color pattern (ayay)D Locus (MLPH)Dark areas of hair and skin are not lightened (DD)B Locus (TYRP1)Black or gray hair and skin (BB)Saddle Tan (RALY)Not expressed (NN)M Locus (PMEL)No merle alleles (mm)
Furnishings (RSPO2) LINKAGELikely unfurnished (no mustache, beard, and/or eyebrows) (II)Coat Length (FGF5)Likely short or mid-length coat (GG)Shedding (MC5R)Likely heavy/seasonal shedding (CC)Coat Texture (KRT71)Likely straight coat (CC)Hairlessness (SGK3)Very unlikely to be hairless (NN)Hairlessness (FOXI3) LINKAGEVery unlikely to be hairless (NN)Oculocutaneous Albinism Type 2 (SLC45A2) LINKAGELikely not albino (NN)
Muzzle Length (BMP3)Likely medium or long muzzle (CC)Tail Length (T)Likely normal-length tail (CC)Hind Dewclaws (LMBR1)Unlikely to have hind dew claws (CC)Blue Eye Color (ALX4) LINKAGELess likely to have blue eyes (NN)Back Muscling & Bulk, Large Breed (ACSL4)Likely normal muscling (CC)
Body Size (IGF1)Larger (NN)Body Size (IGFR1)Larger (GG)Body Size (STC2)Larger (TT)Body Size (GHR - E195K)Larger (GG)Body Size (GHR - P177L)Larger (CC)
Altitude Adaptation (EPAS1)Normal altitude tolerance (GG)
Coefficient Of Inbreeding21%MHC Class II - DLA DRB1Low DiversityMHC Class II - DLA DQA1 and DQB1High Diversity